I am a real person, and have been doing medical research for many years. I opted out of the vaccine due to the red flags. It appeared, early on, that the spike protein was toxic to the cells in the absence of the virus. A study in April 2021 was published by the Salk Institute for Biological Studies:
“In the new study, the researchers created a “pseudovirus” that was surrounded by SARS-CoV-2 classic crown of spike proteins, but did not contain any actual virus. Exposure to this pseudovirus resulted in damage to the lungs and arteries of an animal model—proving that the spike protein alone was enough to cause disease. Tissue samples showed inflammation in endothelial cells lining the pulmonary artery walls.”
https://www.ahajournals.org/doi/10.1161/CIRCRESAHA.121.318902
https://www.salk.edu/news-release/the-novel-coronavirus-spike-protein-plays-additional-key-role-in-illness/
As it turns out, the early reports were right and we now have more information about how the spike 1 protein is destroying our lives.
The synthetic mRNA SARS-CoV-2 produces altered spike 1 protein.
The laboratory manipulated gain-of-function process introduced a nucleotide swap. Uracil in the mRNA molecule was swapped for synthetic methylpseudouracil. The synthetic nucleotide provides two gain-of-function contributions, first by decreasing the human immune reaction to the molecule, and second by increasing the molecular resistance to degradation.
The protein product of the synthetic mRNA is an altered Spike 1 Protein (S1P) coded with two additional proline molecules. These proline molecules provide two additional gain-of-function contributions, first, the proline molecules increase H2 receptor-S1P adhesion, and second, the proline molecules disable the H2 receptors by resisting folding. This results in mis-folded proteins and disabled H2 receptors. The mis-folded proteins set off a cavalcade of mis-folded proteins possibly leading to prion-disease.
The H2 receptor mechanism normally provides protein transportation across the cell membrane.
Spike 1Protein peptides break off and travel through the lymphatic, neurologic, and circulatory systems. The immune system responds by unleashing a storm of proteolytic enzymes, in the form of neutrophilic elastase. Neutrophils are white blood cells with a special proteolytic enzyme that will degrade just about any bacteria, necrotic host tissue, or foreign protein or peptide molecule. Except, the spike 1 protein has a final trick up its sleeve. When the protein particles are broken into smaller peptides, those peptides combine with spike amyloid to form amyloid-like fibrin strands.
The enzyme degradation process produces particles that are able to then reform into amyloid-like fibrils. Studies show that when fibrin is formed in the presence of spike amyloid, the plasmin is unable to break down the fibrin strands, leading to further accumulation of S1P peptide particles. These amyloid-like growths appear to be blocking arteries and veins in the “suddenly deceased”.
There is some data that shows the effectiveness of thiol molecules to inhibit S1P binding to ACE2 receptors. Glutathione and other antioxidant sources of the thiol molecule are abundant in hot water extracted mushrooms. Click on the picture to find the best mushroom growing supplies and mushroom elixirs available!
I'm a retired Clinical Scientist and an Apothecary.
Linet Awolana MBA, BS, MT(AAB)